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Facial canal introitus
Facial canal introitus In anatomy, an introitus is an entrance, one that goes into a canal or hollow organ. The introitus of the facial canal is the entrance to the facial canal, a passage in the temporal bone of the skull through which the facial nerve (the 7th cranial nerve) travels.
RELATED TERMS--------------------------------------
Anatomy The study of form. Gross anatomy involves structures that can be seen with the naked eye. It is as opposed to microscopic anatomy (or histology) which involves structures seen under the microscope.
Organ A structural unit of an animal or plant that serves a specific function.
Temporal Having to do with the side of the head.
Bone Bone refers either to a hardened connective tissue or to one of the individual structures, or organs, into which it is formed, found in many animals. Bones support body structures, protect internal organs, and (in conjunction with muscles) facilitate movement; are also involved with cell formation, calcium metabolism, and mineral storage. The bones of an animal are, collectively, known as the skeleton.
Skull The bony framework of the head.
Nerve Tissue that conveys sensation, temperature, position information to the brain.
Cranial The anatomical term for towards the head; also the general term for of the head. i.e. the lungs are cranial to the pelvis. See Caudal/Inferior/Superior
SIMILAR TERMS--------------------------------------
Facial muscle One of the 43 muscles in the human face. The facial muscles convey basic human emotions such as anger, sadness, fear, surprise, disgust, contempt and happiness by very clear facial signals.
Facing Tooth colored overlay on the visible portion of a crown; may be acrylic, composite or porcelain.
Facioscapulohumeral muscular dystrophy A form of muscular dystrophy that typically begins before age 20 with slowly progressive weakness of the muscles of the face, shoulders, and feet. The severity of the disease is quite variable. Although most people with facioscapulohumeral muscular dystrophy (FSHD) retain the ability to walk, about 20% require a wheelchair. Life expectancy is not shortened. The diagnosis can be confirmed by a DNA test disclosing the deletion of copies of a repeat motif called D4Z4 on chromosome 4. FSHD is inherited in an autosomal dominant manner. Offspring of an affected individual have a 50% chance of inheriting the mutant chromosome 4. About 10-30% of cases are due to a new mutation. Prenatal testing is available.
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Free Radical A destructive fragment of oxygen produced as a byproduct when cells use oxygen to burn fat.
F (coefficient of inbreeding) F is the symbol for the coefficient of inbreeding, a way of gauging how close two people are genetically to one another. The coefficient of inbreeding, F, is the probability that a person with two identical genes received both genes from one ancestor.
F (symbol) A much used symbol, F stands for fractional concentration; free energy; Fahrenheit; visual field; fluorine; force; filial generation, followed by subscript numerals indicating specified matings such as F1); the amino acid phenylalanine; the coefficient of inbreeding , etc.
Facelift A surgical procedure designed to make the face appear younger by pulling loose facial skin taut. With age or excessive sun exposure, wrinkled creased skin can develop on the face, neck or forehead along with fat deposits and folds around the jaws and jowls. While a facelift cannot stop the aging process, it may "turn back the clock" in appearance. Recovery time is usually 1 week, and the results last approximately 10 years. Additional procedures to supplement a facelift -- including necklift, blepharoplasty (eyelid surgery), liposuction, autologous fat injection, removal of buccal (cheek) fat pads, forehead lift, and browlift; chemical or laser peel; and malar (cheek), submalar, or chin implants -- may be necessary to achieve the desired results. Although they are infrequent, risks and complications of facelift surgery include bleeding; hematoma; bruising; infection; neurological dysfunction (loss of muscle function or sensation), which is usually temporary; widened or thickened scars; loss of hair around the incision site; asymmetry (unevenness between two sides); and skin necrosis (loss of skin due to tissue death).
Facelift surgery risks Although infrequent, the risks and complications of facelift surgery include: bleeding, hematoma, bruising; infection; neurological dysfunction (loss of muscle function or sensation), which is usually temporary; widened or thickened scar; loss of hair (around the incision site), asymmetry (unevenness between two sides); and skin necrosis (loss of skin from tissue death).
Facial canal introitus
Facial muscle One of the 43 muscles in the human face. The facial muscles convey basic human emotions such as anger, sadness, fear, surprise, disgust, contempt and happiness by very clear facial signals.
Facioscapulohumeral muscular dystrophy A form of muscular dystrophy that typically begins before age 20 with slowly progressive weakness of the muscles of the face, shoulders, and feet. The severity of the disease is quite variable. Although most people with facioscapulohumeral muscular dystrophy (FSHD) retain the ability to walk, about 20% require a wheelchair. Life expectancy is not shortened. The diagnosis can be confirmed by a DNA test disclosing the deletion of copies of a repeat motif called D4Z4 on chromosome 4. FSHD is inherited in an autosomal dominant manner. Offspring of an affected individual have a 50% chance of inheriting the mutant chromosome 4. About 10-30% of cases are due to a new mutation. Prenatal testing is available.
Factor V A coagulation factor needed for the normal clotting of blood. Also known as proaccelerin.
Factor V Leiden A genetic disorder of blood coagulation (clotting) that carries an increased risk of venous thromboembolism - the formation of clots in veins that may break loose and travel through the bloodstream to the lungs or brain. On the molecular level, factor V Leiden is characterized by a G to A substitution at nucleotide 1691 in the gene for factor V that causes a single amino acid replacement in the factor V protein. On the clotting level, factor V Leiden is inactivated about 10 times slower than normal factor V and persists longer in the circulation, resulting in increased generation of thrombin and a hypercoagulable state (thrombophilia).
Factor VIII Factor eight, a key factor in the process of blood coagulation (clotting). Lack of normal factor VIII causes hemophilia (hemophilia A). The gene for classic hemophilia was long known to be on the X chromosome. Females carry the gene and transmit it to their hemophiliac sons. Female carriers are normal since they have another X chromosome that contains a normal gene to make factor VIII. Each of their sons has a one-half (50:50) risk of being a hemophiliac. Because the gene for hemophilia was clearly on the X chromosome, it was correctly inferred that the normal gene for Factor VIII was on the X. Factor VIII is also known as antihemophiliac factor or antihemophiliac globulin.
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